Chest Breathing | Thoracic Breathing: Effects, Tests and Solutions
Chest breathing (or thoracic breathing) is very common in modern people. More than 50% of adults have predominantly chest breathing at rest. It is even more common for people with chronic diseases, who breathe too deeply at rest, as this table shows.
Minute ventilation rates (chronic diseases)
| All references or
click below for abstracts
|Normal breathing||6 L/min||-||Medical textbooks|
|Healthy Subjects||6-7 L/min||>400||Results of 14 studies|
|Heart disease||15 (~+mn~4) L/min||22||Dimopoulou et al, 2001|
|Heart disease||16 (~+mn~2) L/min||11||Johnson et al, 2000|
|Heart disease||12 (~+mn~3) L/min||132||Fanfulla et al, 1998|
|Heart disease||15 (~+mn~4) L/min||55||Clark et al, 1997|
|Heart disease||13 (~+mn~4) L/min||15||Banning et al, 1995|
|Heart disease||15 (~+mn~4) L/min||88||Clark et al, 1995|
|Heart disease||14 (~+mn~2) L/min||30||Buller et al, 1990|
|Heart disease||16 (~+mn~6) L/min||20||Elborn et al, 1990|
|Pulm hypertension||12 (~+mn~2) L/min||11||D'Alonzo et al, 1987|
|Cancer||12 (~+mn~2) L/min||40||Travers et al, 2008|
|Diabetes||12-17 L/min||26||Bottini et al, 2003|
|Diabetes||15 (~+mn~2) L/min||45||Tantucci et al, 2001|
|Diabetes||12 (~+mn~2) L/min||8||Mancini et al, 1999|
|Diabetes||10-20 L/min||28||Tantucci et al, 1997|
|Diabetes||13 (~+mn~2) L/min||20||Tantucci et al, 1996|
|Asthma||13 (~+mn~2) L/min||16||Chalupa et al, 2004|
|Asthma||15 L/min||8||Johnson et al, 1995|
|Asthma||14 (~+mn~6) L/min||39||Bowler et al, 1998|
|Asthma||13 (~+mn~4) L/min||17||Kassabian et al, 1982|
|Asthma||12 L/min||101||McFadden, Lyons, 1968|
|COPD||14 (~+mn~2) L/min||12||Palange et al, 2001|
|COPD||12 (~+mn~2) L/min||10||Sinderby et al, 2001|
|COPD||14 L/min||3||Stulbarg et al, 2001|
|Sleep apnea||15 (~+mn~3) L/min||20||Radwan et al, 2001|
|Liver cirrhosis||11-18 L/min||24||Epstein et al, 1998|
|Hyperthyroidism||15 (~+mn~1) L/min||42||Kahaly, 1998|
|Cystic fibrosis||15 L/min||15||Fauroux et al, 2006|
|Cystic fibrosis||10 L/min||11||Browning et al, 1990|
|Cystic fibrosis*||10 L/min||10||Ward et al, 1999|
|CF and diabetes*||10 L/min||7||Ward et al, 1999|
|Cystic fibrosis||16 L/min||7||Dodd et al, 2006|
|Cystic fibrosis||18 L/min||9||McKone et al, 2005|
|Cystic fibrosis*||13 (~+mn~2) L/min||10||Bell et al, 1996|
|Cystic fibrosis||11-14 L/min||6||Tepper et al, 1983|
|Epilepsy||13 L/min||12||Esquivel et al, 1991|
|CHV||13 (~+mn~2) L/min||134||Han et al, 1997|
|Panic disorder||12 (~+mn~5) L/min||12||Pain et al, 1991|
|Bipolar disorder||11 (~+mn~2) L/min||16||MacKinnon et al, 2007|
|Dystrophia myotonica||16 (~+mn~4) L/min||12||Clague et al, 1994|
Note that advanced stages of asthma can lead to lung destruction, ventilation-perfusion mismatch,
and arterial hypercapnia causing further reduction in body oxygen levels.
More than 90% of sick people have upper chest breathing with increased minute ventilation, respiratory rates, and minute volume (i.e., automatic deep breathing at rest or taking too much air per one breath). Thoracic breathing causes three fundamental health effects that promote chronic diseases and lead to low body-oxygen levels.
Chest breathing reduces blood oxygenation
The textbook, Respiratory Physiology (West, 2000), suggests that the lower 10% of the lungs transports more than 40 ml of oxygen per minute, while the upper 10% of the lungs transports less than 6 ml of oxygen per minute. Hence, the lower parts of the lungs are about 6-7 times more effective in oxygen transport than the top of the lungs due to richer blood supply mostly caused by gravity.
During thoracic breathing, lower layers of the lungs, which are most valuable in oxygen transport, get much less, if any, fresh air (less oxygen supply). This causes reduced oxygenation of arterial blood in the lungs and can lead to so called "ventilation-perfusion" mismatch (as in COPD or emphysema). Normal breathing is diaphragmatic, allowing homogeneous inflation of both lungs with fresh air, similar to what happens in the cylinder of a car engine due to the movement of the piston. Hence, during diaphragmatic breathing, all alveoli are homogeneously stretched vertically and get fresh air supply with higher O2 concentration for superior arterial blood oxygenation. In contrast, chest breathing creates problems with blood oxygenation. This leads to reduced cell oxygenation: the driving force of all chronic diseases.
Thoracic breathing causes lymphatic stagnation
Dr. Shields, in his study, "Lymph, lymph glands, and homeostasis" (Shields, 1992) reported that diaphragmatic breathing stimulates the cleansing work of the lymph system by creating a negative pressure pulling the lymph through the lymph system. This increases the rate of elimination of toxins from visceral organs by about 15 times. Why is this so?
The lymph system, unlike the cardiovascular system with the heart, has no pump. Lymph nodes are located in parts of the human body that get naturally compressed (squeezing) due to movements of body parts. For example, lymph nodes are located around the neck, above arm pits and groin area. Hence, when we move, stretch or turn the head, arms and legs, these lymph nodes get mechanical stimulation to push the lymph through valves. This is how the lymphatic system works. However, the lymph nodes connected to the stomach, kidneys, liver, pancreas, spleen, large and small colons, and other vital organs are located just under the diaphragm - over 60% of all lymph nodes in total!
Hence, nature expects us to use the diaphragm in order to remove waste products from these vital organs all the time - literally with each breath, 24/7. Hence, another problem with thoracic breathing is stagnation in the lymph system and accumulation of waste products in vital organs located under the diaphragm. (This effect is also mentioned in other sources, for example, http://www.amsa.org/healingthehealer/breathing.cfm.)
Thoracic breathing means low blood oxygen
People who are chest breathers virtually always have deep breathing (large breaths) at rest or sleep and suffer from hyperventilation (breathing more than the norm). When we breathe more air, we get less oxygen in body cells. In fact, the slower your automatic breathing pattern at rest (down to only 3 breaths/min), the larger the amount of oxygen delivered to cells.
Keep in mind that, while healthy normal breathing is abdominal or diaphragmatic. It is very small in amount (only 500 ml of air per one breath at rest) so that healthy people usually do not feel their breath.
Find your type of breathing at rest
Do you breathe using the diaphragm or chest at rest? Check yourself.
Easy test. Put one hand on your abdomen (or stomach) and another one higher, on your upper chest (see the picture on the right). Relax completely so that your breathing dynamic has little changes. Pay attention to your breathing for about 20-30 seconds with both hands in place. (You want to know more about your usual unconscious breathing and find out if you have chest or abdominal breathing.) Take 2-3 very slow and deep breaths to feel your breathing dynamics in more detail.
Now you know more about your usual breathing pattern. In order to be certain, you can ask other people to observe how you breathe when you are not aware of your breathing (e.g., during sleep, while reading, studying, etc.).
Learn how to stop chest breathing
Module 8 (Learning Section). How to Learn and Develop Diaphragmatic Breathing 24/7 with 3 breathing exercises, instructions, techniques, and long term solutions to thoracic breathing problems.
This video clip (Chest Breathing in Modern People) explains why modern people are chest breathers:
Reference pages: Breathing norms and the DIY body oxygen test:
- Breathing norms: Parameters, graph, and description of the normal breathing pattern
- Body-oxygen test (CP test) : How to measure your own breathing and body oxygenation (two in one) using a simple DIY test
References: pages about CO2 effect:
- Vasodilation: CO2 expands arteries and arterioles facilitating perfusion (or blood supply) to all vital organs
- The Bohr effect: How and why oxygen is released by red blood cells in body tissues
- Nerve stabilization: Carbon dioxide has powerful calmative and sedative effects on brain neurons and nerve cells
Castro M. Control of breathing. In: Physiology, Berne RM, Levy MN (editors), 4-th edition, Mosby, St. Louis, 1998.
Ganong WF, Review of medical physiology, 15-th ed., 1995, Prentice Hall Int., London.
Shields JW, MD, Lymph, lymph glands, and homeostasis, Lymphology, Dec. 1992, 25, 4: 147.
West JB. Respiratory physiology: the essentials. 6th ed. Philadelphia: Lippincott, Williams and Wilkins; 2000.
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