Treat Causes of Heart Disease Naturally
disease (or cardiovascular disease) is a common medical term for a variety of diseases
that affect the heart. Official medicine claims that there are no known causes and
no successful treatments for heart disesase. Currently, up
to about one quarter of people die due to heart disease
in leading Western countries. It is the main cause of
mortality in the USA, Canada, the UK and many other countries.
Coronary heart disease is the main type of
heart disease. It is a disorder of the blood vessels of the heart that can
lead to a heart attack. A heart attack happens when an artery becomes too
constricted (due to vasoconstriction) or blocked, preventing blood, oxygen
and nutrient supply to the cells of the heart muscle. Heart disease is
only one of several other cardiovascular diseases, such as stroke, angina
(chest pain), hypertension (high blood pressure), and rheumatic heart
What causes cardiovascular diseases?
The cause of heart disease is simple. Consider this graph and table.
If you click on the image, you can see a web page with a table that includes
exact details and medical studies that discovered heavy breathing in people
with cardiovascular disease.
These studies prove that people with heart disease breathe much more air at rest than
the medical norms. As a result, they have too low levels of CO2 in the lungs and
arterial blood. CO2 is the most potent known dilator of blood vessels.
Therefore, people with heart disease have reduced blood and oxygen supply to
their heart muscles due to their heavy breathing (chronic hyperventilation).
How to treat cardiovascular diseases naturally: experience of over 180 MDs
Western official medicine wrongly claims that heart disease is impossible to treat and reverse.
Stop Heart Attack, Prevent
Cardiovascular Disease. Based on the clinical
experience and medical statistical data of over 180 Russian medical doctors, we can
positively assert that chronic heart disease can be reversed. This is possible since it is
based on an ineffective breathing pattern (over-breathing or hyperventilation),
which decreases heart oxygenation.
How can hyperventilation affect the heart muscle and cardiovascular system?
Many effects resulting from a CO2 deficiency can influence cardiovascular disease.
* Low blood CO2 values lead to the narrowing of small
blood vessels (arteries and arterioles) in the whole body (see Webpage: CO2 effects -
Vasodilation; all links are below). This causes two problems. First, blood
flow to the heart muscle decreases (Okazaki et al, 1991). Hence, the heart tissue
gets less oxygen, glucose and other nutrients. Second, since small blood vessels
are the main contributors to the total resistance in relation to blood flow, CO2
deficiency increases resistance to blood flow and makes the work of the heart
more difficult leading to an increased heart rate or pulse.
* The suppressed Bohr effect, due to low CO2 values in the arterial blood,
also reduces oxygenation of the heart muscle (Webpage: CO2 effects - The Bohr effect).
This increases anaerobic metabolism and
produces excessive amounts of lactic acid producing more stress on the heart. Note that lactic acid is often
implicated as a source of pain in any tissue. In the case of the heart, a person
can suffer from angina or chest pain.
* These 2 effects reduce oxygen levels in the brain, heart and all
other vital organs. Angina pain is caused by one factor: low oxygenation of
the heart tissue.
* The excited nerve cells in the heart (the cells that are called pacemakers)
interfere with the normal synchronization and harmony in the working of the
heart muscle (see Webpage: CO2 effects - Stabilization of the nervous system).
Indeed, the heart valves should open and close in proper time, much like a
well-tuned engine. Desynchronization can make the whole process of blood pumping
less efficient or more energy- and oxygen-demanding.
More cardiovascular disease factors discovered by Russian Buteyko doctors
* Dr. Buteyko and other Buteyko breathing medical doctors conducted medical studies
devoted to heart disease and cholesterol levels. Hyperventilation causes
abnormal metabolism of fats. This leads to
increased blood cholesterol level in predisposed people. That condition gradually, over
periods of weeks or months, produces cholesterol deposits on the walls of blood
vessels in genetically predisposed people. Such deposits can induce primary
hypertension. As Russian published studies suggest, the CP has a linear
correlation with the blood cholesterol level.
* Chronic hyperventilation affects the normal work of essential fatty acids
causing changes in inflammatory responses and the malfunctioning of the immune
* Mouth breathing (both at rest and during exercise) is an additional adverse
stimulus. It prevents the normal absorption of nitric oxide (a hormone and powerful
dilator of blood vessels) synthesized in the nasal passages.
The father of cardiorespiratory physiology, Yale University Professor Yandell
Henderson (1873-1944), investigated some of these effects about a century ago.
Among his numerous physiological studies, he performed experiments with
anaesthetized dogs on mechanical ventilation. The results were described in his
publication Acapnia and shock. - I. Carbon dioxide as a factor in the regulation
of the heart rate. In this article, published in 1908 in the American Journal of
Physiology, he wrote, “... we were enabled to regulate the heart to any desired
rate from 40 or fewer up to 200 or more beats per minute. The method was very
simple. It depended on the manipulation of the hand bellows with which
artificial respiration was administered... As the pulmonary ventilation
increased or diminished, the heart rate was correspondingly accelerated or
retarded” (p.127, Henderson, 1908).
* Since heart patients breathe 2-3 times more than the official norm, they
usually have chest breathing, which reduces blood oxygenation. These
irregularities indicate abnormal states of the autonomous nervous and musculoskeletal systems.
How could these factors lead to different forms of cardiovascular disease?
whole picture depends on genetic predisposition and environmental factors. There
are so many negative factors created by hyperventilation. They can affect the normal work of
any part of the cardiovascular
system. People are different. Some may get chronic heart failure, others high
blood pressure, or stroke, atrial fibrillation, or various other abnormalities in the heart muscle.
Do you know that it is possible to get an abnormal ECG tracing from a healthy
heart just by voluntary heavy breathing? Later, many cardiac professionals,
while analyzing such ECGs, can claim pathological changes in the heart causing cardiovascular disease. These
changes are different in different people. Vice versa, normal breathing
naturally eliminates various, already detected, ECG abnormalities.
How more than 180 Russian Buteyko MDs reverse cardiovascular disease
medicine and physiology have very limited understanding of what is
going on with the cardiovascular system when breathing and CO2 values gradually
change in one direction or another. There are many questions related to
individual variability, mechanisms of developing pathologies, the interaction of
hereditary and environmental factors.
However, Russian and Soviet Buteyko medical
professionals found that patients with heart disease need to slow down their
automatic breathing: breathe less and less, day after day. This increases
body oxygen levels. Patients with less than 10 seconds for the body oxygen
test have severe forms of the heart disease. About 15 seconds corresponds to
moderate heart problems and 20 s to light forms of cardiovascular disease.
Over 40 seconds for the body-oxygen test prevents or
quickly reverses any heart problems, even abnormalities in the heart muscle
tissue. The simple DIY body-oxygen test is a very sensitive parameter of the
degree of heart disease.
Note that many heart patients, when they have less than 20 seconds for the
body-oxygen test, cannot practice breathing exercises that include breath holds.
They can find more information about Restrictions, limits,
and temporary contraindications for heart disease during the early stages of
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