Chronic Inflammation Causes and Natural Treatment
Do you suffer from constant inflammation on the face or in the sinuses, throat, lower back, muscles, stomach, or other body parts? Chronic inflammation, according to new research, is a process that is
essentially controlled by the electric potential (abundance or deficiency of free
electrons) of the organism. What has been taught and known for decades is an
artificial scenario that models the situation based on our modern abnormal
In our current understanding, inflammation is a response of the immune system to injury. As a part of this response, the immune system sends white blood cells to the site of injury. These white cells include neutrophils that produce an oxidative burst in the injured area. The neutrophils release reactive oxygen- and reactive nitrogen species (also known as free radicals) in order to effectively destroy pathogens (bacteria) that usually penetrate across the skin into the human body. These free radicals also break apart damaged cells so as to rebuild healthy tissue and ensure healing. This process of destruction is based on the chemical aggressiveness of free radicals based on their ability to "steal" electrons from other molecules.
Now we come to the main problem that causes chronic inflammation. Together with the destruction of damaged cells, free radicals also leak into surrounding areas and destroy healthy cells causing the classic quintet of hallmarks of inflammation: PRISH or Pain, Redness, Immobility (loss of function), Swelling and Heat. However, this (abnormal) scenario takes place in modern humans (and during animal studies) only in conditions of electrical insulation from Earth and results from electron deficiency.
Body electricity and chronic inflammation
Humans, just several generations ago, used to be electrically grounded to Earth (due to barefoot life and
absence of artificial fabrics) for nearly 24/7 just several generations ago, and
this provided the human body with a slightly negative electrical charge that
corresponds to Earth's negative potential. However, nearly all modern research is performed on
insulated humans and animals who have a positive charge or electron deficiency.
What are the effects? Let us consider the key problem associated with
Grounding the human body results in deactivation of free radicals in healthy
tissues since Earth can provide an abundant supply of electrons to neutralize
free radicals, prevent damage of healthy cells, and "quench"
chronic inflammation. As a result, grounding,
within 10-30 minutes, reduces chronic inflammation and pain. Here is a link to one of the
studies that provide references and thermal images related to effects of grounding
Therefore, the first and easy step in order to reduce chronic inflammation is to provide
free electrons for the body or ensure those natural conditions that existed during human
and animal evolution.
However, this is not the end of the story. Chronic inflammation
also requires low blood supply and reduced O2 levels in tissues (cell hypoxia).
Main cause of chronic or constant inflammation
We see that sick people are affected by chronic overbreathing that leads to
tissue hypoxia (regardless of the ventilation-perfusion mismatch and CO2 levels
in the arterial blood). Among other factors associated with chronic inflammation, according to recent research studies, are
pro-inflammatory transcription factors, such as nuclear factor kappa B
(NF-kappaB), activator protein (AP)-1
(Safronova & Morita, 2010; Ryan et al, 2009), and hypoxia-inducible
factor 1 (Imtiyaz & Simon, 2010; Sumbayev & Nicholas, 2010).
Therefore, hyperventilation (or breathing more than the medical norms) is an additional effect and cause of
in modern people.
Constant Inflammation: Natural Treatment
In order to reduce chronic inflammation, we need to breathe slower and less
24/7. Why is this
so? We need more oxygen in tissues to normalize key physiological processes and eliminate symptoms of chronic diseases and chronic
inflammation. Just Earthing is not enough.
Hence, fast and effective treatment of chronic inflammation is based on:
- grounding yourself by standing on Earth barefoot or using methods for grounding during sleep
and work (here are more details for practical steps: Earthing)
- restoration of normal breathing parameters in order to increase body
oxygenation and normalize chief physiological parameters.
Furthermore, prevention and avoidance of allergic reactions leading to chronic inflammation are necessary for the clinical remission of chronic inflammation. Upon achievement of
about 35-40 s for the body-oxygen test (this corresponds
to the medical norm for breathing), different types of chronic
inflammation disappear within 2-3 weeks or even faster. This is true for asthma,
bronchitis, gastritis, sinusitis, pancreatitis, duodenitis, hepatitis, arthritis, problems with liver, and many
others conditions. Numerous students completely eliminated their inflammation on the face, in the throat, sinuses, stomach, lower back, muscles and other body parts. Furthermore, Russian Buteyko breathing doctors had a
successful clinical trial on patients with liver cirrhosis and hepatitis B, while thousands
of their patients solved problems with
chronic inflammation using breathing retraining.
Laffey JG, Kavanagh BP, Carbon dioxide and the critically ill--too little of a good thing?, Lancet. 1999 Oct 9;354(9186):1283-6.
...hypocapnia is associated with many acute illnesses (eg, asthma, systemic inflammatory response syndrome...
Hanidziar D, Koulmanda M, Inflammation and the balance of Treg and Th17 cells in transplant
rejection and tolerance., Curr Opin Organ Transplant. 2010 Aug;15(4):411-5.
...Inflammation of the allograft, occurring as a
consequence of hypoxia and ischemia/reperfusion injury...
Imtiyaz HZ, Simon MC, Hypoxia-inducible factors as essential regulators of inflammation, Curr Top Microbiol Immunol. 2010;345:105-20.
...Myeloid cells provide important functions in low oxygen (O(2)) environments created by pathophysiological conditions, including sites of infection, inflammation...
Sumbayev VV, Nicholas SA, Hypoxia-inducible factor 1 as one of the "signaling drivers" of Toll-like receptor-dependent and allergic inflammation., Arch Immunol Ther Exp (Warsz). 2010 Aug;58(4):287-94. Epub 2010 May 26.
Hypoxia-inducible factor 1 (HIF-1) is a heterodimeric transcription
complex which plays a crucial role in cellular adaptation to low oxygen
Tkacova R, Systemic inflammation in chronic obstructive pulmonary disease: may adipose tissue play a role? Review of the literature and future perspectives., Mediators Inflamm. 2010;2010:585989. Epub 2010 Apr 20.
...obesity-related hypoxia results in local inflammatory response within adipose tissue per se...
Safronova O, Morita I, Transcriptome remodeling in hypoxic inflammation., J Dent Res. 2010 May;89(5):430-44. Epub 2010 Mar 26.
...This review summarizes the current knowledge on hypoxia-responsive transcriptional pathways in inflammation and their importance in the etiology of chronic inflammatory diseases...
Eltzschig HK, Rivera-Nieves J, Colgan SP, Targeting the A2B adenosine receptor during gastrointestinal ischemia and inflammation., Expert Opin Ther Targets. 2009 Nov;13(11):1267-77.
...In addition, we discuss the role of this pathway in dampening hypoxia-elicited inflammation, specifically in the setting of intestinal ischemia and inflammation.
Ryan S, Taylor CT, McNicholas WT, Systemic inflammation: a key factor in the pathogenesis of
cardiovascular complications in obstructive sleep apnoea syndrome?, Thorax. 2009 Jul;64(7):631-6.
...Intermittent hypoxia, the hallmark of OSAS, results in activation of pro-inflammatory transcription factors...
Chao J, Wood JG, Gonzalez NC, Alveolar hypoxia, alveolar macrophages, and systemic inflammation., Respir Res. 2009 Jun 22;10:54.
... alveolar macrophages activated by hypoxia release mediators into the circulation. This mediators ... initiate a widespread systemic inflammation.
Garvey JF, Taylor CT, McNicholas WT, Cardiovascular disease in obstructive sleep apnoea syndrome: the
role of intermittent hypoxia and inflammation., Eur Respir J. 2009 May;33(5):1195-205.
...There is increasing evidence that intermittent hypoxia plays a role
in the development of cardiovascular risk in obstructive sleep apnoea
syndrome (OSAS) through the activation of inflammatory pathways...
Arnaud C, Dematteis M, Pepin JL, Baguet JP, Levy P, Obstructive sleep apnea, immuno-inflammation, and atherosclerosis., Semin Immunopathol. 2009 Jun;31(1):113-25. Epub 2009 Apr 29.
...This IH (intermittent hypoxia)induces several consequences such as hemodynamic, hormonometabolic, oxidative, and immuno-inflammatory alterations...
Oliver KM, Taylor CT, Cummins EP, Hypoxia. Regulation of NFkappaB signalling during inflammation: the
role of hydroxylases., Arthritis Res Ther. 2009;11(1):215. Epub 2009 Feb 23.
...Microenvironmental hypoxia has long been identified as being coincident with chronic inflammation...
Ramalho R, Guimaraes C, [The role of adipose tissue and macrophages in chronic inflammation
associated with obesity: clinical implications] [Article in Portuguese], Acta Med Port. 2008 Sep-Oct;21(5):489-96. Epub 2009 Jan 16.
...so, hypoxia can be a critical factor in inflammatory obese state manifestation...
Taylor CT, Interdependent roles for hypoxia inducible factor and nuclear factor-kappaB in hypoxic inflammation., J Physiol. 2008 Sep 1;586(Pt 17):4055-9. Epub 2008 Jul 3.
...hypoxia impacts upon the development of inflammation through the coordinated expression of adaptive, inflammatory and apoptotic genes...
Frede S, Berchner-Pfannschmidt U, Fandrey J, Regulation of hypoxia-inducible factors during inflammation., Methods Enzymol. 2007;435:405-19.
...The microenvironment of inflamed and injured tissue is characterized by low levels of oxygen and glucose and high levels of inflammatory cytokines....
Wouters EF, Local and systemic inflammation in chronic obstructive pulmonary
disease., Proc Am Thorac Soc. 2005;2(1):26-33.
...The main causes of systemic inflammation in COPD remain to be elucidated, although systemic hypoxia is a candidate factor...
Joussen AM, Fauser S, Krohne TU, Lemmen KD, Lang GE, Kirchhof B, Diabetic retinopathy. Pathophysiology and therapy of hypoxia-induced
inflammation [Article in German], Ophthalmologe. 2003 May;100(5):363-70.
New breathing students with terminal conditions (end-stage disease) are accepted on CureEndStageDisease.com with Dr. Artour's Triple Guarantee.
You can leave your grammatically correct feedback and/or comment on the most relevant page. It may be below. Thanks.