Cough Treatment: 3 Easy Breathing Exercises
This most natural cough-treatment method (a simple breathing exercise) was developed and used by over 170 Russian medical doctors. The breathing exercise has been tested on thousands of patients with asthma, cystic fibrosis, and COPD with a striking success. Over 90% of people can dramatically reduce their bouts of coughing.
Tens of medical studies proved that people with asthma, COPD, bronchitis, and many other "coughers" breathe about 2-3 times more than the medical norm. Overbreathing makes any cough treatment ineffective unless it is eliminated.
Ventilation rates (chronic diseases)
| All references or
click below for abstracts
|Normal breathing||6 L/min||-||Medical textbooks|
|Healthy Subjects||6-7 L/min||>400||Results of 14 studies|
|Asthma||13 (~+mn~2) L/min||16||Chalupa et al, 2004|
|Asthma||15 L/min||8||Johnson et al, 1995|
|Asthma||14 (~+mn~6) L/min||39||Bowler et al, 1998|
|Asthma||13 (~+mn~4) L/min||17||Kassabian et al, 1982|
|Asthma||12 L/min||101||McFadden, Lyons, 1968|
|COPD||14 (~+mn~2) L/min||12||Palange et al, 2001|
|COPD||12 (~+mn~2) L/min||10||Sinderby et al, 2001|
|COPD||14 L/min||3||Stulbarg et al, 2001|
|Cystic fibrosis||15 L/min||15||Fauroux et al, 2006|
|Cystic fibrosis||10 L/min||11||Browning et al, 1990|
|Cystic fibrosis*||10 L/min||10||Ward et al, 1999|
|CF and diabetes*||10 L/min||7||Ward et al, 1999|
|Cystic fibrosis||16 L/min||7||Dodd et al, 2006|
|Cystic fibrosis||18 L/min||9||McKone et al, 2005|
|Cystic fibrosis*||13 (~+mn~2) L/min||10||Bell et al, 1996|
|Cystic fibrosis||11-14 L/min||6||Tepper et al, 1983|
are the effects of chronic heavy breathing in relation to a possible treatment of cough?
Among the known effects of chronic hyperventilation and hypocapnia (see CO2
links in resources), in relation to
- overstimulation or irritation of cough receptors in the tracheobronchial tree and larynx due to hypocapnia (low CO2)
- constriction of airways due to hypocapnia
- mechanical irritation of the constricted airways due to large movements of air during coughing
- chronic respiratory infections and inflammation due to suppression of the immune system caused by cell hypoxia and other effects caused by hyperventilation
- low oxygen content in mitochondria of cilia cells that prevent normal removal of mucus and other debris out of the lungs and airways (not only humans, cilia cells also experience chronic fatigue due to low O2 content, causing their inability to beat in unison and remove mucus and pathogens).
Complete and best treatment of coughing
Here are details for this best and successful cough treatment. This YouTube video (below) is currently ranked as Number 1 for "cough treatment" in YouTube search.
If you retrain your breathing pattern so that you have 30 or more seconds for the body-oxygen test 24/7, your chronic problems with coughing will disappear completely.
Related web pages:
- Get Rid of Cough - The main breathing exercise that is used during bouts of coughing to stop them and reduce the damage caused by coughing to airways, lungs, and all body cells
- Stop Coughing At Night - A breathing technique and exercise to reduce duration and severity of night coughing. A similar exercise is used for insomnia problems
- Cause of Cough - A general overview of the physiological mechanism that makes coughing chronic or persistent.
J Assoc Physicians India. 2000 Mar; vol 48(no. 3): p. 343-345.
The role of cough and hyperventilation in perpetuating airway inflammation in asthma.
Singh V, Chowdhary R, Chowdhary N.
Department of Pulmonary Medicine, SMS Medical College, Jaipur-302 016, India.
Air flowing through a pipe exerts frictional stress on the walls of the pipe. Frictional stress of more than 40 N/m2 (velocity equivalent of air 113 m/s) is known to cause acute endothelial damage in blood vessels. The frictional stress in airways during coughing may be much greater, however, since the velocity of air may be as high as speed of sound in air. We suggest that high levels of frictional stress perpetuate airway inflammation in airways which are already inflamed and vulnerable to frictional stress-induced trauma in patients with asthma. Activities associated with rapid ventilation and higher frictional stress (e.g. exercise, hyperventilation, coughing, sneezing and laughing) cause asthma to worsen whilst activities that reduce frictional stress (Yoga 'Pranayama', breathing a helium-oxygen mixture and nasal continuous positive airway pressure) are beneficial. Therefore control of cough may have anti-inflammatory benefits in patients with asthma.
Respir Physiol Neurobiol. 2007 Jun 15; 156(3): p. 331-339.
Cough and ventilatory adjustments evoked by aerosolised capsaicin and distilled water (fog) in man.
Lavorini F, Pantaleo T, Geri P, Mutolo D, Pistolesi M, Fontana GA.
Dipartimento di Area Critica Medico Chirurgica, UnitÓ Funzionale di Medicina Respiratoria, UniversitÓ di Firenze, Viale G.B. Morgagni 85, 50134 Firenze, Italy.
Airway receptors mediate cough and ventilatory adjustments. Simultaneous assessment of cough sensory-motor components and changes in breathing pattern may provide insights into the receptors prevailingly stimulated by inhaled irritants. Nineteen subjects inhaled capsaicin and fog up to threshold concentrations for cough. Cough intensity, respiratory sensations and changes in breathing pattern induced by the two irritants were compared. Capsaicin and fog cough threshold values did not correlate. Coughing induced by both agents was preceded by qualitatively similar sensations and by significant increases in minute ventilation and respiratory drive due to selective increases in tidal volume (P<0.01). Cough intensity was similar with both agents. Cough frequency and the intensity of the urge to cough were higher with capsaicin (P<0.01). The lack of correlation between fog and capsaicin cough threshold values suggests differences in the neural mechanisms activated. The selective increase in tidal volume suggests prevailing involvement of rapidly adapting receptors. The stronger sensations evoked by capsaicin may contribute to the higher cough frequency observed with this agent.
Monaldi Arch Chest Dis. 1999 Jun;54(3):275-9.
Advances in understanding and treatment of cough.
Sherrington School of Physiology, St. Thomas' Hospital Campus (UMDS), London, UK.
Many different conditions and diseases cause cough. The commonest acute causes are pollution, including cigarette smoke, and upper respiratory tract infection. The commonest chronic causes are postnasal drip, asthma, chronic bronchitis and gastro-oesophageal reflux. Epidemiological studies give widely different patterns of incidence. The different conditions that cause cough have in common the fact that the cough is mediated via the vagus nerves, with sensory receptors in and under the epithelium from the larynx down to the smaller bronchi. These receptors are polymodal, responding to a large variety of stimuli, including mechanical and chemical irritants, inflammatory mediators, intraluminal material and large volume changes of the lungs. With irritation and inflammation, C fibre receptors release neurokinins such as substance P, which in turn stimulate cough receptors. The central nervous pathways for the cough reflex are poorly understood. They can be activated or inhibited voluntarily. Studies on the pharmacology of the central nervous pathways of coughing are opening up new therapeutic possibilities. Other new therapies include drugs acting on the sensory receptors for cough, thereby avoiding adverse central nervous effects.
Pulm Pharmacol Ther. 2007;20(4):416-22.
The problem of cough and development of novel antitussives.
Department of Thoracic Medicine, National Heart and Lung Institute, Dovehouse Street, London SW3 6LY, UK. firstname.lastname@example.org
Cough is a very common clinical symptom and current therapies are largely ineffective, indicating a major unmet medial need. There is a pressing need to develop novel and safe antitussive therapies. This is likely to arise from better understanding of the sensory nerves involved in cough and the signalling pathways that are activated. A major therapeutic target should be sensitization of the cough reflex which is a feature of patients with both acute (virally induced) cough and chronic cough, including chronic idiopathic cough. Studies on human cough mechanisms are limited. There are several novel therapeutic approaches that are currently being explored. Perhaps the most promising drugs are transient receptor potential vanilloid-1 (TRPV(1)) antagonists, selective cannabinoid agonists (CB2 agonists), maxi-K channel openers and P2X3 antagonists. New cough therapies may target airway nerve sensitization and may best be delivered as inhalers to minimize any systemic effects. Understanding the intercellular signalling pathways involved in nociception may lead to novel drugs, such as p38 mitogen-activated protein (MAP) kinase inhibitors, being used in the treatment of cough in the future. It is also likely that several novel treatments that are developed as analgesics will also prove to be beneficial in the treatment of cough.
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