Most Natural Cough Treatment
This most natural cough
treatment therapy has been tested on thousands of people with respiratory disorders,
mostly asthma. It is probably the only
treatment program for cough problems that has clear understanding of
the cause of chronic cough and physiological effects and mechanism of
persistent coughing. As a part of this medical treatment, there is a simple
breathing exercise that has a powerful immediate therapeutic effect in
many situations, including whooping cough (or wet cough), dry cough (or
chesty cough), coughing at night, coughing in children and many others.
The links to this and other exercises are provided at the bottom of
this page.
Persistent or chronic cough results in overbreathing. Tens of medical studies proved that people with asthma, COPD, bronchitis, and many other "coughers" breathe about 2-3 times more than the medical norm (see the links to these studies below).
What
are the effects?
Among the known effects of chronic hyperventilation and hypocapnia (see CO2
links in resources), in relation to
cough, are:
- overstimulation or irritable state of cough receptors in the
tracheobronchial tree and larynx due to hypocapnia (low CO2)
- constriction of airways due to hypocapnia
- mechanical irritation of the constricted airways due to large
movements of air during coughing
- chronic respiratory infections and inflammation due to suppression of
the immune system caused by cell hypoxia and other effects caused by
hyperventilation
- low oxygen content in mitochondria of cilia cells that prevent normal
removal of mucus and other debris out of the lungs and airways (not
only humans, cilia cells also experience chronic fatigue due to low O2
content causing their inability to beat in unison and remove mucus and
pathogens).
If you retrain your breathing pattern so that you have 25 or more
seconds for the body oxygen test, your
chronic problems with coughing (including wet or whooping cough, chest or dry
cough, coughing at night, etc.) will disappear completely
(provided that you avoid triggers of your allergies).
Related web pages
Get Rid of Cough - The main
breathing exercise that is used during bouts of coughing to stop them and reduce the damage caused by coughing to airways, lungs, and
all body cells
How to Stop Coughing At Night -
A breathing technique and exercise to reduce duration and severity of
night coughing. A similar exercise is used for insomnia problems.
The Cause of Cough - A general
overview of the physiological mechanism that makes coughing chronic or
persistent.
Reference Web Pages: Breathing norms, Medical Graphs and Tables about Breathing Rates (Minute Ventilation) and
Body Oxygen in Healthy, Normal and Sick People
Breathing
norms Parameters, graph, and description of the normal
breathing pattern
6 breathing myths 6
myths about breathing and body oxygenation (prevalence: over 90%)
Hyperventilation Definitions of
hyperventilation: their advantages and weak points
Hyperventilation Syndrome in the
Sick. Table
1. Western scientific evidence about prevalence of CHV
(chronic hyperventilation) in patients with various chronic conditions
(34 medical studies)
Normal Minute Ventilation in
Healthy Subjects: Easy and Light Breathing (14 Studies)
Hyperventilation Prevalence Present in Over 90% of
Normal People (24 medical publications)
HV and hypoxia
How and why deep breathing reduces oxygenation of cells and tissues of
all vital organs
Body oxygen test
How to measure your own breathing and body oxygenation (a simple DIY test)
Body oxygen in healthy
Table 4. CP (body oxygen level) in healthy people (27 medical
studies)
Body oxygen in sick Table 5.
CP (body oxygen level) in sick people (14 medical studies)
Buteyko
Table of Health Zones with clinical description of most common zones
Morning HV Morning
hyperventilation effect or how and why critically ill people are most
likely to die during early morning hours
References: CO2 Effects Web Pages
Vasodilation: CO2 expands arteries and arterioles facilitating perfusion
(or blood
supply) to all vital organs
The Bohr effect
How and why oxygen is released by red blood cells in tissues
Cell Oxygen Levels and oxygen transport are controlled by
alveolar CO2 and breathing
Oxygen Transport depends on
breathing and these two effects (Vasoconstriction-Vasodilation and the Bohr
effect) are parts of two diagrams that summarize influences of hypocapnia (low CO2
content in the blood and cells) on circulation and O2 delivery
Free Radical Generation takes
place due to anaerobic cell respiration caused by cell hypoxia. Hence,
antioxidant defenses of the human body are also regulated by CO2 and breathing
Inflammatory Response is controlled by
breathing since hypoxia leads to or intensifies chronic inflammation through over-expression
of the hypoxia-inducible factor 1, while normal
breathing reduces these processes
Nerve stabilization takes place due to calmative or
sedative effects of carbon dioxide in neurons or nerve cells
Muscle relaxation or relaxation of muscle cells
is normal at high CO2, while hypocapnia causes muscular tension, poor posture
and, sometimes, aggression and violence
Brochodilation - dilation of
airways (bronchi and bronchioles) by carbon dioxide, and their constriction due
to hypocapnia
CO2: Best Natural Cough Suppressant
and "home remedy" since it calms urge-to-cough nerve receptors located in the
tracheobronchial tree and larynx
Blood
pH regulation and regulation of other bodily fluids
CO2: Lung Damage Healer: Elevated carbon
dioxide prevents injury and promotes healing of lung tissues
CO2: Skin and Tissue Healer
Synthesis of Glutamine
in the Brain, CO2 fixation, and other chemical reactions
CO2 myth
"CO2 is a toxic waste gas" myth
Breathing control
How is our breathing regulated? Why hypocapnia makes breathing uneven and erratic?
References
J Assoc Physicians India. 2000 Mar; vol 48(no. 3): p. 343-345.
The role of cough and hyperventilation in perpetuating airway
inflammation in asthma.
Singh V, Chowdhary R, Chowdhary N.
Department of Pulmonary Medicine, SMS Medical College, Jaipur-302 016,
India.
Air flowing through a pipe exerts frictional stress on the walls of the
pipe. Frictional stress of more than 40 N/m2 (velocity equivalent of
air 113 m/s) is known to cause acute endothelial damage in blood
vessels. The frictional stress in airways during coughing may be much
greater, however, since the velocity of air may be as high as speed of
sound in air. We suggest that high levels of frictional stress
perpetuate airway inflammation in airways which are already inflamed
and vulnerable to frictional stress-induced trauma in patients with
asthma. Activities associated with rapid ventilation and higher
frictional stress (e.g. exercise, hyperventilation, coughing, sneezing
and laughing) cause asthma to worsen whilst activities that reduce
frictional stress (Yoga 'Pranayama', breathing a helium-oxygen mixture
and nasal continuous positive airway pressure) are beneficial. Therefore
control of cough may have anti-inflammatory benefits in patients with
asthma.
Respir Physiol Neurobiol. 2007 Jun 15; 156(3): p. 331-339.
Cough and ventilatory adjustments evoked by aerosolised capsaicin and
distilled water (fog) in man.
Lavorini F, Pantaleo T, Geri P, Mutolo D, Pistolesi M, Fontana GA.
Dipartimento di Area Critica Medico Chirurgica, Unitą Funzionale di
Medicina Respiratoria, Universitą di Firenze, Viale G.B. Morgagni 85,
50134 Firenze, Italy.
Airway receptors mediate cough and ventilatory adjustments.
Simultaneous assessment of cough sensory-motor components and changes
in breathing pattern may provide insights into the receptors
prevailingly stimulated by inhaled irritants. Nineteen subjects
inhaled capsaicin and fog up to threshold concentrations for cough.
Cough intensity, respiratory sensations and changes in breathing
pattern induced by the two irritants were compared. Capsaicin and fog
cough threshold values did not correlate. Coughing induced by both
agents was preceded by qualitatively similar sensations and by
significant increases in minute ventilation and respiratory drive due
to selective increases in tidal volume (P<0.01). Cough intensity was
similar with both agents. Cough frequency and the intensity of the urge
to cough were higher with capsaicin (P<0.01). The lack of
correlation between fog and capsaicin cough threshold values suggests
differences in the neural mechanisms activated. The selective increase
in tidal volume suggests prevailing involvement of rapidly adapting
receptors. The stronger sensations evoked by capsaicin may contribute
to the higher cough frequency observed with this agent.
Monaldi Arch Chest Dis. 1999 Jun;54(3):275-9.
Advances in understanding and treatment of cough.
Widdicombe JG.
Sherrington School of Physiology, St. Thomas' Hospital Campus (UMDS),
London, UK.
Many different conditions and diseases cause cough. The commonest acute
causes are pollution, including cigarette smoke, and upper respiratory
tract infection. The commonest chronic causes are postnasal drip,
asthma, chronic bronchitis and gastro-oesophageal reflux.
Epidemiological studies give widely different patterns of incidence. The
different conditions that cause cough have in common the fact that the
cough is mediated via the vagus nerves, with sensory receptors in and
under the epithelium from the larynx down to the smaller bronchi. These
receptors are polymodal, responding to a large variety of stimuli,
including mechanical and chemical irritants, inflammatory mediators,
intraluminal material and large volume changes of the lungs. With
irritation and inflammation, C fibre receptors release neurokinins such
as substance P, which in turn stimulate cough receptors. The central
nervous pathways for the cough reflex are poorly understood. They
can be activated or inhibited voluntarily. Studies on the pharmacology
of the central nervous pathways of coughing are opening up new
therapeutic possibilities. Other new therapies include drugs acting on
the sensory receptors for cough, thereby avoiding adverse central
nervous effects.
Pulm Pharmacol Ther. 2007;20(4):416-22.
The problem of cough and development of novel antitussives.
Barnes PJ.
Department of Thoracic Medicine, National Heart and Lung Institute,
Dovehouse Street, London SW3 6LY, UK. p.j.barnes@imperial.ac.uk
Cough is a very common clinical symptom and current therapies are
largely ineffective, indicating a major unmet medial need. There is
a pressing need to develop novel and safe antitussive therapies. This
is likely to arise from better understanding of the sensory nerves
involved in cough and the signalling pathways that are activated. A
major therapeutic target should be sensitization of the cough reflex
which is a feature of patients with both acute (virally induced) cough
and chronic cough, including chronic idiopathic cough. Studies on human
cough mechanisms are limited. There are several novel therapeutic
approaches that are currently being explored. Perhaps the most
promising drugs are transient receptor potential vanilloid-1 (TRPV(1))
antagonists, selective cannabinoid agonists (CB2 agonists), maxi-K
channel openers and P2X3 antagonists. New cough therapies may target
airway nerve sensitization and may best be delivered as inhalers to
minimize any systemic effects. Understanding the intercellular
signalling pathways involved in nociception may lead to novel drugs,
such as p38 mitogen-activated protein (MAP) kinase inhibitors, being
used in the treatment of cough in the future. It is also likely that
several novel treatments that are developed as analgesics will also
prove to be beneficial in the treatment of cough.
Back to Symptoms of Hyperventilation
| Disclaimer | Copyright © 2011 Artour Rakhimov | About Artour | Contributions | Contact details | Promote this site |
